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Anti-HDAC9 antibody

SKU Product Brand Unit Availability Price Quantity  
AB-06-2116
Anti-HDAC9 antibody
Erpan Tech In stock

Specifications        

Product Cat#: AB-06-2116
Product type: Primary antibody
Antigen: HDAC9
Immunogen: Purified recombinant fragment of human HDAC9 expressed in E. Coli.
Species immunized: Mouse
Isotype: IgG1
Applications: Western Blot (1:200-1:2000); Flow Cytometry (1:100-1:200)
Reactivity: Human
Clonality (clone number): Monoclonal (C4-B7)
Form: Liquid
Buffer: 1*TBS (pH7.4), 1%BSA, 40%Glycerol. Preservative: 0.05% Sodium Azide.
Concentration: 1 mg/ml
Purity: ProA affinity purified
Storage: Aliquot and freeze at -20℃. Avoid multiple freeze/thaw cycles.
Alternative names: HD 7 antibody
HD 7B antibody
HD 9 antibody
HD7 antibody
HD7B antibody
HD9 antibody
HDAC 7 antibody
HDAC 7B antibody
HDAC 9 antibody
HDAC 9B antibody
HDAC 9FL antibody
HDAC antibody
HDAC7B antibody
HDAC9 antibody
HDAC9_HUMAN antibody
HDAC9B antibody
HDAC9FL antibody
HDRP antibody
Histone deacetylase 4/5 related protein antibody
Histone deacetylase 7 antibody
Histone deacetylase 7B antibody
Histone deacetylase 9 antibody
Histone deacetylase 9A antibody
Histone deacetylase related protein antibody
Histone deacetylase-related protein antibody
KIAA0744 antibody
MEF2 interacting transcription repressor MITR antibody
MEF2 interacting transcription repressor protein antibody
MEF2-interacting transcription repressor MITR antibody
MITR antibody
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Target information

Responsible for the deacetylation of lysine residues on the N-terminal part of the core histones (H2A, H2B, H3 and H4). Histone deacetylation gives a tag for epigenetic repression and plays an important role in transcriptional regulation, cell cycle progression and developmental events. Represses MEF2-dependent transcription.Isoform 3 lacks active site residues and therefore is catalytically inactive. Represses MEF2-dependent transcription by recruiting HDAC1 and/or HDAC3. Seems to inhibit skeletal myogenesis and to be involved in heart development. Protects neurons from apoptosis, both by inhibiting JUN phosphorylation by MAPK10 and by repressing JUN transcription via HDAC1 recruitment to JUN promoter.

Provider

Erpantech Laboratory

download

MSDS-AB-06-2116.pdf (180 downloads )